-IBIS-1.7.6-
tx
cutaneous system
burn
Nutrition
dietary guidelines
therapeutic foods:
foods rich in Vitamins A, E and C
foods rich in Silicon (Jensen)
for serious burns, add fresh vegetable and fruit juices or other foods rich in electrolytes: Potassium, Sodium, Calcium, Phosphorous
specific remedies:
apply honey topically to minor burns
mix 2 oz. beancurd and l oz. raw brown sugar and apply topically to minor burns (Butt and Bloomfield, 124.)
apply raw potato juice to affected parts (Chao-liang, Qing-rong, Bao-zhen, 14.)
drink the liquid obtained by steaming fresh yellow soybeans (Chao-liang, Qing-rong, Bao-zhen, 71.)
supplements
Vitamin C, 1,000-2,000 mg, three times per day, to enhance tissue healing and support immune function
Vitamin B-complex
Vitamin A 50,000 IU per day
Beta-carotene, 100,000 IU per day, to enhance tissue healing and support immune function
Vitamin E, 1200 IU d-alpha tocopherols, three times daily, for anti-oxidant function and to reduce scarring; it is important to use the tocopherol form of vitamin E when treating burns topically as it is immediately available.
Potassium and other electrolytes
Zinc, 30 mg, twice daily, to enhance tissue healing and support immune function
Bromelain, 250 to 500 mg, four times daily, to decrease inflammation.
Essential fatty acids, safflower oil 2 Tbsp per day
L-glutamine, 4-10 g, three times daily, provides amino acid to nourish intestinal mucosa and protect against damage secondary to severe burns.
» drug interaction:
Iron, Sodium, Calcium, carotene, Vitamin B12, fat, protein, cholesterol and neomycin (Mycifradin, Neobiotic): neomycin damages the intestinal villi and precipitates bile salts in the lumen; this leads to malabsorption of the enumerated nutrients
Vitamin K and cephalosporins [cefuroxime (Ceftin), cephalexin (Cefanex, Keflet, Keflex, Keftab), cefixime (Suprax), cephadroxil (Duricef, Ultracef), cephaclor (Ceclor)]:
- cephalosporins interfere with Vitamin K metabolism, sometimes leading to hemmorhage
- cephalosporins reduce Vitamin K synthesis by destroying the colonic bacteria responsible for that process
footnotes